peripheral resistance การใช้

• It reduces blood pressure and peripheral resistance and produces fluid retention.
• Peripheral resistance is determined by functional and anatomic changes in small arteries and arterioles.
• Many mechanisms have been proposed to account for the rise in peripheral resistance in hypertension.
• Exposure to vascular hypertrophy, leading to progressive increases in peripheral resistance and blood pressure.
• The drug acts on the renin-angiotensin system to decrease total peripheral resistance in two ways.
• Nitric oxide reduces both total peripheral resistance and venous return, thus decreasing both preload and afterload.
• This relaxation will decrease total peripheral resistance, which will in turn decrease venous return to the heart.
• This will result in a decrease in total peripheral resistance and a decrease in preload on the heart.
• Its actions are to increase peripheral resistance via end-tidal CO 2 or E T CO 2 levels.
• Vasodilation directly affects the relationship between mean arterial pressure, cardiac output, and total peripheral resistance ( TPR ).
• Normal EABV exists when the ratio of cardiac output to peripheral resistance maintains venous return and cardiac output at normal levels.
• Hypertension increases the afterload, since the LV has to work harder to overcome the elevated arterial peripheral resistance and decreased compliance.
• It has also been suggested that endothelial dysfunction and vascular inflammation may also contribute to increased peripheral resistance and vascular damage in hypertension.
• The increased peripheral resistance and greater blood volume place further strain on the heart and accelerates the process of damage to the myocardium.
• Systolic, diastolic, pulse and mean arterial pressures and total peripheral resistance were significantly lower in the DOW group than in the control group.
• This is associated with increased stroke volume of the left ventricle and decrease in the peripheral resistance leading to the widened pulse pressure of aortic regurgitation.
• By coupling Sympathetic inhibition leads to a drop in peripheral resistance, while parasympathetic activation leads to a depressed heart rate ( reflex bradycardia ) and contractility.
• Furthermore, this increased cardiac output is coupled with lowered total peripheral resistance; meaning, while the heart is pumping faster, the vasculature is relaxed.
• Carpaine, 2 mg / kg, reduced cardiac output, stroke volume, stroke work, and cardiac power, but the calculated total peripheral resistance remained unchanged.
• It is a vasodilator of coronary and peripheral vessels, which reduces peripheral resistance and afterload, though not as potent as the dihydropyridine ( DHP ) calcium channel blockers.